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Cartalax research peptide, ≥98% purity by HPLC, CAS N/A (Khavinson bioregulator), lyophilized powder for in-vitro laboratory research, Pure Peptides
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Supplied as a lyophilized powder and independently verified to ≥98% purity by HPLC and MS-UPLC analysis.

333.31 g/mol≥98% (HPLC)N/A (Khavinson bioregulator)

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What is Cartalax?

Cartalax is a synthetic three-amino-acid peptide (Ala-Glu-Asp) from the Russian peptide bioregulator program, designed to target cartilage and connective tissue. It is studied in laboratory research for its effects on cartilage-cell signaling.

  • A short synthetic peptide (Ala-Glu-Asp) from the Russian bioregulator program
  • Studied in laboratory research for its effects on cartilage and connective-tissue cell signaling
  • One of a family of short peptides each studied in a specific tissue type
  • Investigated alongside other organ-specific bioregulators from the same program

For research use only. Not approved for human therapeutic use.

Cartalax, also known by its tripeptide designation AED (Ala-Glu-Asp) and the developmental code T-31, is a synthetic tripeptide bioregulator with the molecular formula C12H19N3O8 and an approximate molecular weight of 333.3 g/mol. Developed at the Saint Petersburg Institute of Bioregulation and Gerontology, Cartalax was designed based on peptide fractions associated with cartilaginous tissue. Its three-amino acid sequence, Ala-Glu-Asp, shares the AED core motif found in the longer tetrapeptide bioregulators Epithalon (AEDG), Cortagen (AEDP), Cardiogen (AEDR), and Bronchogen (AEDL), representing the minimal tripeptide unit within this structural series. Produced via solid-phase peptide synthesis, Cartalax is associated with chondrocyte gene expression and cartilage matrix signaling pathways.

Cartalax is a tripeptide bioregulator within the Khavinson framework. In mesenchymal stem cell preparations, the tripeptide modulated the expression of genes governing chondrogenic differentiation and cellular metabolic activity, consistent with the Wnt, ERK-p38, and Smad signaling pathways described for chondrogenic peptides [1]. In a parallel in vitro model of mesenchymal stem cell aging in culture, AED was reported to increase IGF1 gene expression approximately 3.5- to 5.6-fold alongside stimulation of NFκB gene expression in the same model (FOXO1 and TNKS2 effects were reported for the related peptides KED and KE, not AED) [2]. At the whole-animal level, in aged rats, AED administration was associated with reduced proteinuria and increased diuresis [3], making Cartalax a reference compound in preclinical connective-tissue and cellular-aging research examining chondrogenic differentiation, regulation of IGF1 and associated gene expression, and morphofunctional changes in aged tissue.

Cartalax is produced to research-grade standards and independently verified by third-party HPLC and MS-UPLC analysis before dispatch. Vials are vacuum sealed and stored in a temperature controlled, monitored cold storage system. Certificates of Analysis are available to view and download directly on the product page.

Sold strictly for in vitro research purposes only. Not for human consumption. Intended for use by qualified researchers in laboratory settings only.

Scientific Review

Dr. Martina Rossi, PhD

Dr. Martina Rossi, PhD

Scientific Contributor and Reviewer

Reviewed for scientific accuracy, 14 June 2026

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